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Heavy drinking-even for just a few days at a time-can cause fat to build up in the liver. This condition, called steatosis, or fatty liver, is the earliest stage of alcoholic liver disease and the most common alcohol-induced liver disorder. The excessive fat makes it more difficult for the liver to operate and leaves it open to developing dangerous inflammations, like alcoholic hepatitis.

For some, alcoholic hepatitis does not present obvious symptoms. For others, though, alcoholic hepatitis can cause fever, nausea, appetite loss, abdominal pain, and even mental confusion. As it increases in severity, alcoholic hepatitis dangerously enlarges the liver, and causes jaundice, excessive bleeding, and clotting difficulties.

Another liver condition associated with heavy drinking is fibrosis, which causes scar tissue to build up in the liver. Alcohol alters the chemicals in the liver needed to break down and remove this scar tissue. As a result, liver function suffers.

If you continue to drink, this excessive scar tissue builds up and creates a condition called cirrhosis, which is a slow deterioration of the liver. Cirrhosis prevents the liver from performing critical functions, including managing infections, removing harmful substances from the blood, and absorbing nutrients.

A variety of complications, including jaundice, insulin resistance and type 2 diabetes, and even liver cancer, can result as cirrhosis weakens liver function.

Risk factors ranging from genetics and gender, to alcohol accessibility, social customs around drinking, and even diet can affect a person's individual susceptibility to alcoholic liver disease. Statistics show that about one in five heavy drinkers will develop alcoholic hepatitis, while one in four will develop cirrhosis.


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